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Three Researchers To Receive Prestigious Awards From The American Society Of Hematology

�The American Society of
Hematology (ASH), the world's largest professional society of blood
specialists, will honour three scientists who bear made significant
contributions to the savvy of hematologic diseases.


At the 2008 ASH Annual Meeting, Clara D. Bloomfield, M.D., of the Ohio
State University in Columbus, OH, will be presented with the Henry M.
Stratton Medal, intended to honor an individual whose well-recognized
contributions to haematology have taken place over a stop of several
years. Dr. Bloomfield will receive this award for her noteworthy
achievements in the surface area of hematologic malignancies, specially acute
myelogenous leukemia, for more than three decades. Dr Bloomfield has been a
major contributor to the savvy of the biology of these diseases and
the practical use of biologic information in diagnosis, classification, and
determinant prognosis and selection of curative therapeutical approaches.


Kenneth Anderson, M.D., of the Dana-Farber Cancer Institute in Boston,
MA, volition be presented with the William Dameshek Prize, awarded to an
individual world Health Organization has made a late outstanding contribution to the field. Dr.
Anderson will be recognized for his contributions to the treatment of
myeloma. He has advance the field by establishing a new paradigm focussed
not merely on the malignant cell, but too on the microenvironment for the
designation of molecularly-targeted therapies. His rapid translations of
his pre-clinical discoveries into form I-III trials have considerably
improved the clinical final result for myeloma patients.


Robert Kyle, M.D., of the Mayo Clinic in Rochester, MN, will be
presented with the Wallace H. Coulter Award for Lifetime Achievement in
Hematology, which was established in 2007 to honor individuals who receive
made a lifelong committedness to the specialty and whose contributions have
had a major impact on education, research, and/or practice. Dr. Kyle will
receive the laurels for his contributions to the field of study of multiple myeloma,
monoclonal antibody gammopathies, amyloidosis, and related to plasma-cell disorders.
Throughout his 50-year career as a physician-researcher, educator, and
consultant, he has focused on defining these diseases, understanding their
pathogenesis, demonstration, and prognosis, and design and evaluating
therapeutic approaches. In add-on, he has authored more than a thousand
publications has trained more than 200 practicing hematologists.


These awards are named after notable figures in the Society's history
and testament be awarded during the 50th ASH Annual Meeting in December at the
Moscone Center in San Francisco. Dr. Kyle will be presented with his laurels
prior to the Plenary Scientific Session on Sunday, December 7, at 1:30 p.m.
EST. Drs. Bloomfield and Anderson will be presented with their awards at
the Presidential Symposium to be held on Tuesday, December 9, at 9:30 a.m.
For the complete annual meeting schedule and additional information, please
visit hypertext transfer protocol://www.haematology.org/meetings/2008.


The American Society of Hematology is the world's
largest professional

Hunger Hormone May Protect Against Stress Induced Depression And Anxiety

�By doing tests on mice federal official on a calorie restricted diet, US researchers take in discovered that ghrelin, a hormone that increases when people
don't eat, may defend against symptoms of depression or anxiety brought on by stress.




The research is the work of scientists lED by aged author Dr Jeffrey Zigman, assistant prof of inner medicine and psychiatry at the University of Texas (UT) Southwestern Medical Center in Dallas. It was published on-line on 15th June
2008 in the journal Nature Neuroscience.




"Our findings in mice suggest that chronic stress causes ghrelin levels to go up and that behaviors associated with depression and anxiety decrease
when ghrelin levels rise," aforesaid Zigman, adding that:




"An unfortunate side result, however, is increased food intake and body weight."




Lead author and instructor of psychiatry at UT Southwestern, Dr Michael Lutter, said:




"Our findings support the idea that these hunger hormones don't do just unitary thing; quite, they align an entire behavioral response to tenseness and
in all probability affect modality, stress and energy levels."




Scientists like Zigman and colleagues already knew that fasting causes the gut to produce ghrelin, which is involved in sending hunger signals to the
mental capacity, and this has lED them to suggest blocking ghrelin as an approach to weight control.




But this new subject shows that if you do that, it could interfere with the body's natural way of dealing with anxiety and natural depression.




For the study, Zigman and his team commit "wild type" (ie not genetically altered in whatever way) mice on a calorie restricted diet for 10 days, which lED to a
four-fold increase in their ghrelin levels. Stress tests involving mazes and forced swims showed that the calorie restricted mice had reduced
symptoms of anxiety and depression compared to control couterparts that had been allowed unrestricted access to food.




The researchers then bred genetically neutered mice whose bodies did not respond to ghrelin and federal official them a calorie restricted diet as well, but under
tenseness tests their depression and anxiety symptoms stayed high; there was no anti-depression or anti-anxiety effect as there had been in the dotty type
mice on the low nutritionist's calorie diet.




Finally, Zigman and colleagues did a third prove, this time to explore social tenseness where normal wild type and genetically altered mice were exposed to aggressive "bully" mice every mean solar day (an access often used to study parallels of depression in humans).




Under stress atmospheric condition both types of mice had raised ghrelin that lasted for up to four weeks after existence bullied for the final time, only the mice that had been genetically altered not to respond to gherlin showed more social shunning behaviour and ate less than the non-altered mice, suggesting they had more depression symptoms.





A possible explanation could be the survival reward that is gained from such an effect over countless generations of evolution where the main
priority is getting enough nutrient to avoid starving to death. Zigman suggested that hunter gatherers needed to remain becalm in order of magnitude to hunt
successfully (or they could end being someone else's meal). Perhaps ghrelin induced by hunger leading to anti-anxiety gave them the edge over
their competitors.




These findings might also explain what happens in conditions like anorexia nervosa aforementioned Lutter:




"We're very interested to see whether ghrelin discourse could help people with anorexia nervosa, with the idea organism that in a certain population,
calorie restriction and weight release could make an antidepressant effect and could be reinforcing for this illness."




Zigman, Lutter and colleagues bob Hope to continue with their research and look at the areas of the brain where ghrelin could be causation these
antidepressant-like effects.



"The orexigenic hormone ghrelin defends against depressive symptoms of continuing stress."


Michael Lutter, Ichiro Sakata, Sherri Osborne-Lawrence, Sherry A Rovinsky, Jason G Anderson, Saendy Jung, Shari Birnbaum, Masashi Yanagisawa,
Joel K Elmquist, Eric J Nestler & Jeffrey M Zigman

Nature Neuroscience Published online 15 June 2008.


DOI: 10.1038/nn.2139



Click here for
Abstract.



Sources: Nature Neuroscience press liberation and abstract.




Written by: Catharine Paddock, PhD



Copyright: Medical News Today


Not to be reproduced without permit of Medical News Today



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